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A new full-scale crash simulator is being employed in a biomechanics research program for establishing quantitative specifications for human tolerance to impact forces. Intact, fully articulated human cadavers are used as test subjects. Design capacity of the facility is a 40 mph barrier-type collision. A test sled equipped with load cell supported impact targets and carrying a seated cadaver subject is accelerated pneumatically, released, and arrested at collision-level decelerations. Impact forces at the knee and chest are recorded, and X-ray surveillance for skeletal damage is conducted. In a current study of instrument-panel type knee-impact situations, normal femurs in embalmed male cadavers 50–75 years of age fractured at loads of 1500 pounds and greater.

Sternal and/or spinal acceleration data from eighteen of the unembalmed cadaver, blunt thoracic impact experiments reported by the authors at the Eighteenth Stapp Conference have been analyzed and several related response parameters computed. High acceleration levels, rates of onset, and Gadd Severity Indices were found in sternal acceleration measurements, whereas quite low values were obtained from the spinal regions in the same tests. A Severity Index value of 1000, computed from a sternal acceleration measurement, would be associated with only a mild exposure; whereas for a spinal measurement, the same value would reflect an extremely severe exposure. Correlation matrices which include cadaver characteristics, input and response parameters, and Abbreviated Injury Scale ratings show that none of the sternal acceleration parameters correlate well with AIS rating in the analyzed data base.

The purpose of this paper is to present a comparison of whole body, target impingement knee impact response for a Part 572 dummy versus that for anthropometrically similar embalmed human cadavers. “Response” is defined here to include the impact force-time history as sensed by 1) femur load cells, and 2) impingement target load cells for the dummy and by the target load cells for the cadavers. The data presented demonstrate significantly higher peak forces and correspondingly shorter pulse durations for the dummy than for the companion cadaver subjects under similar test conditions and at all velocity levels investigated. For the dummy, the ratio of forces measured by the femur load cells to those measured by the impingement target load cells averaged eight tenths.

Thoracic impact response and injuries of living and postmortem porcine siblings were investigated to quantify comparative differences. Thirteen male animals, averaging 61.4 kg, from five different porcine litters comprised the two animal samples. Porcine brothers were subjected to similar impact exposures for which at least one brother was tested live, anesthetized and another dead, post rigor with vascular repressurization. Statistically significant differences in biomechanical responses and injuries were observed between live and postmortem siblings. On the average the anesthetized live animals demonstrated a greater thoracic compliance, as measured by increased normalized total deflections (21% Hi), and reduced overall injuries (AIS 14% Lo and rib fractures 26% Lo) at lower peak force levels (13% Lo) than did the postmortem subjects. However, individual comparisons of “match-tested” siblings demonstrated very similar responses in some cases.

Details of a new crash simulator and preliminary results from a series of cadaver knee impact experiments were presented at the Eighth Stapp Conference. During the past year additional data concerning injury to the knee-thigh-hip complex have been obtained, and the studies have been extended to consider impact to the chest. Results to date indicate that for knee impacts against a moderately padded surface it is not possible to predict whether failure of the patella, femur or pelvis will occur first, although in these studies femoral fractures occurred most frequently. A force of 1400 lb. is recommended at this time as a reasonably conservative value for the over-all injury threshold level. Volunteers tolerated impact loads to the knee of 800-1000 lb. For loads applied over the sternum through a 25-30 padded surface, static and dynamic thoracic stiffness characteristics were determined for a limited number of cadavers.

At the 1970 SAE International Automobile Safety Conference, the first experimental chest impact results from a new, continuing biomechanics research program were presented and compared with earlier studies performed elsewhere by one of the authors using a different technique. In this paper, additional work from the current program is documented. The general objective remains unchanged: To provide improved quantification of injury tolerance and thoracic mechanical response (force-time, deflection-time, and force-deflection relationships) for blunt sternal impact to the human cadaver. Fourteen additional unembalmed specimens of both sexes (ranging in age from 19-81 years, in weight from 117-180 lb, and in stature from 5 ft 1-1/2 in to 6 ft) have been exposed to midsternal, blunt impacts using a horizontal, elastic-cord propelled striker mass. Impact velocities were higher than those of the previous work, ranging from 14-32 mph.

Previous studies of human thoracic injury tolerance and mechanical response to blunt, midsternal, anteroposterior impact loading were reported by the authors at the 1970 SAE International Automobile Safety Conference and at the Fifteenth Stapp Car Crash Conference. The present paper documents additional studies from this continuing research program and provides an expansion and refinement of the data base established by the earlier work. Twenty-three additional unembalmed cadavers were tested using basically the same equipment and procedures reported previously, but for which new combinations of impactor mass and velocity were used in addition to supplementing other data already presented. Specifically, the 43 lb/11 mph (19.5 kg/4.9m/s) and 51 lb/16 mph (23.1 kg/7.2 m/s) conditions were intercrossed and data obtained at 43 lb/16 mph (19.5 kg/7.2 m/s) and 51 lb/11 mph (23.1 kg/4.9 m/s).

As part of a continuing study of thoracic injury resulting from blunt frontal loading, the interrelationship of velocity and chest compression was investigated in a series of animal experiments. Anesthetized male swine were suspended in their natural posture and subjected to midsternal, ventrodorsad impact. Twelve animals were struck at a velocity of 14.5 ± 0.9 m/s and experienced a controlled thoracic compression of either 15, 19, or 24%. Six others were impacted at 9.7 ± 1.3 m/s with a greater mean compression of 27%. For the 14.5 m/s exposures the severity of trauma increased with increasing compression, ranging from minor to fatal. Injuries included skeletal fractures, pulmonary contusions, and cardiovascular ruptures leading to tamponade and hemothorax. Serious cardiac arrhythmias also occurred, including one case of lethal ventricular fibrillation. The 9.7 m/s exposures produced mainly pulmonary contusion, ranging in severity from moderate to critical.

Results of two studies concerning the interrelationship of velocity, compression and injury in blunt thoracic impact to anesthetized swine have been combined to provide a data base of forty-one experiments. impact velocity ranged from ∼8-30 m/s and applied normalized chest compression from ∼0.10-0.30. Experimental subjects were suspended in the spine-horizontal position and loaded midsternally through a 150 mm diameter, flat rigid disk on an impacting mass propelled upward from below. Measurements and computations included sternal and spinal accelerations, intracardiovascular overpressures, physiological responses, injury, as assessed by necropsy, and different forms of the velocity and compression exposure severity parameters. The significance of both compression and velocity as parameters of impact exposure severity is clearly demonstrated. Qualitatively, exacerbation of injury was seen when either variable was increased with the other held constant.

The influence of body posture, and inherently support, on thoracic impact response was investigated in an animal model. Anesthetized and postmortem domestic swine were exposed to blunt, midsternal loading while supported in their natural quadrupedal posture, and the results were compared with previously reported data from similar tests involving an upright body orientation. Twelve male animals were tested, six while anesthetized and six postmortem. Each animal was impacted once by a 21 kg rigid mass with a flat contact interface moving at a nominal velocity of either 8 or 10 m/s. Measured mechanical responses included applied load, sternal and spinal accelerations, thoracic compression and aortic overpressure. Injury response was assessed from a thoracico-abdominal necropsy. In addition, ECG traces were recorded pre and postimpact to monitor electro-physiological response.

Currently used criteria based on functions of spinal acceleration obtained from crash test dummies are shown to be invalid indicators of chest injuries in blunt frontal impacts. Cadaver impact data are analyzed; and injury is found to be a statistically significant function of chest deflection, chest depth, and cadaver age at death. Based on the resulting regression equations, injury-limiting chest deflections are recommended for various size test dummies. The recommendations apply only to test dummies that have significant thoracic biofidelity for blunt frontal impact. They are valid for environments which include signigicant blunt frontal impact. Their extension to other environments has not been validated.

Far-side lateral impacts were simulated using a Part 572 dummy and human cadavers to compare responses for several belt restraint configurations. Sled tests were conducted having a velocity change of 35 km/hr at a 10 g deceleration level. It was estimated from field data that a 35 km/hr velocity change of the laterally struck vehicle represents about an 80th percentile level for injury-producing lateral collisions. Subjects restrained by a three-point belt system with an outboard anchored diagonal shoulder belt (i.e., positioned over the shoulder opposite the side of impact) rotated out of the shoulder belt and onto the seat. The subject received some lateral restraint due to interaction with the shoulder belt and seatback. The subjects restrained by a three-point belt system with an inboard anchored diagonal shoulder belt (i.e., positioned over the shoulder on the side of impact) remained essentially upright due to shoulder belt interaction with the neck and/or head.

Seventeen left lateral impact experiments were performed using anesthetized swine to determine the biomechanics of injury production in this impact mode. Two series of eight animals were used and one animal served as a control. In the first series of experiments, rigid thoracic and pelvic loading surfaces were separated by an “interplate gap” of 20.3 cm (8”). In the second series of experiments, the interplate gap was filled by a rigid plate mounted flush with the thoracic and pelvic loading surfaces. Impact velocities ranged from 7.2 to 15.0 m/s (about 15 to 30 mph). Injury patterns for the liver, spleen, and rib cage were significantly different in the two series of experiments (level of significance > 90%). The causative factor responsible for the different injury outcomes was the interplate gap. The conclusion of this report is that loading-surface discontinuities can cause significant injury.