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This study evaluated the response of restrained post-mortem human subjects (PMHS) in 40 km/h frontal sled tests. Eight male PMHS were restrained on a rigid planar seat by a custom 3-point shoulder and lap belt. A video motion tracking system measured three-dimensional trajectories of multiple skeletal sites on the torso allowing quantification of ribcage deformation. Anterior and superior displacement of the lower ribcage may have contributed to sternal fractures occurring early in the event, at displacement levels below those typically considered injurious, suggesting that fracture risk is not fully described by traditional definitions of chest deformation. The methodology presented here produced novel kinematic data that will be useful in developing biofidelic human models.

Rear seat adult occupant protection is receiving increased attention from the automotive safety community. Recent anthropomorphic test device (ATD) studies have suggested that it may be possible to improve kinematics and reduce injuries to rear seat occupants in frontal collisions by incorporating shoulder-belt force-limiting and pretensioning (FL+PT) technologies into rear seat 3-point belt restraints. This study seeks to further investigate the feasibility and potential kinematic benefits of a FL+PT rear seat, 3-point belt restraint system in a series of 48 kmh frontal impact sled tests (20 g, 80 ms sled acceleration pulse) performed with post mortem human surrogates (PMHS). Three PMHS were tested with a 3-point belt restraint with a progressive (two-stage) force limiting and pretensioning retractor in a sled buck representing the rear seat occupant environment of a 2004 mid-sized sedan.

Very little experimental research has focused on the kinematics, dynamics, and injuries of rear-seated occupants. This study seeks to develop a baseline response for rear-seated post mortem human surrogates (PMHS) in frontal crashes. Three PMHS sled tests were performed in a sled buck designed to represent the interior rear-seat compartment of a contemporary midsized sedan. All occupants were positioned in the right-rear passenger seat and subjected to simulated frontal crashes with an impact speed of 48 km/h. The subjects were restrained by a standard, rear seat, 3-point seat belt. The response of each subject was evaluated in terms of whole-body kinematics, dynamics, and injury. All the PMHS experienced excessive forward translation of the pelvis resulting in a backward rotation of the torso at the time of maximum forward excursion.

Traumatic aortic rupture (TAR) accounts for a significant mortality in automobile crashes. A numerical method by means of a mesh-based code coupling is employed to elucidate the injury mechanism of TAR. The aorta is modeled as a single-layered thick wall composed of two families of collagen fibers using an anisotropic strain energy function with consideration of viscoelasticity. A set of constitutive parameters is identified from experimental data of the human aorta, providing strict local convexity. An in vitro aorta model reconstructed from the Visible Human dataset is applied to the pulsatile blood flow to establish the references of mechanical quantities for physiological conditions. A series of simulations is performed using the parameterized impact pulses obtained from frontal sled tests.

Injury to the thorax is the predominant cause of fatalities in crash-involved automobile occupants over the age of 65, and many elderly-occupant automobile fatalities occur in crashes below compliance or consumer information test speeds. As the average age of the automotive population increases, thoracic injury prevention in lower severity crashes will play an increasingly important role in automobile safety. This study presents the results of a series of sled tests to investigate the thoracic deformation, kinematic, and injury responses of belted post-mortem human surrogates (PMHS, average age 44 years) and frontal anthropomorphic test devices (ATDs) in low-speed frontal crashes. Nine 29 km/h (three PMHS, three Hybrid III 50th% male ATD, three THOR-NT ATD) and three 38 km/h (one PMHS, two Hybrid III) frontal sled tests were performed to simulate an occupant seated in the right front passenger seat of a mid-sized sedan restrained with a standard (not force-limited) 3-point seatbelt.

Rupture of the thoracic aorta is a leading cause of rapid fatality in automobile crashes, but the mechanism of this injury remains unknown. One commonly postulated mechanism is a differential motion of the aortic arch relative to the heart and its neighboring vessels caused by high-magnitude acceleration of the thorax. Recent Indy car crash data show, however, that humans can withstand accelerations exceeding 100 g with no injury to the thoracic vasculature. This paper presents a method to investigate the efficacy of acceleration as an aortic injury mechanism using high-acceleration, low chest deflection sled tests. The repeatability and predictability of the test method was evaluated using two Hybrid III tests and two tests with cadaver subjects. The cadaver tests resulted in sustained mid-spine accelerations of up to 80 g for 20 ms with peak mid-spine accelerations of up to 175 g, and maximum chest deflections lower than 11% of the total chest depth.

This paper presents a computational study of the effects of three parameters on the resulting thoracic injury criteria in side impacts. The parameters evaluated are a) door velocity-time (V-t) profile, b) door interior padding modulus, and c) initial door-to-occupant offset. Regardless of pad modulus, initial offset, or the criterion used to assess injury, higher peak door velocity is shown to correspond with more severe injury. Injury outcome is not, however, found to be sensitive to the door velocity at the time of first occupant contact. A larger initial offset generally is found to result in lower injury, even when the larger offset results in a higher door velocity at occupant contact, because the increased offset results in contact later in the door V-t profile - closer to the point at which the door velocity begins to decrease. Cases of contradictory injury criteria trends are identified, particularly in response to changes in the pad modulus.

This paper presents an analysis of the displacement measurement of the Hybrid III 50th percentile male dummy chest in quasistatic and dynamic loading environments. In this dummy, the sternal chest deformation is typically characterized using a sliding chest potentiometer, originally designed to measure inward deflection in the central axis of the dummy chest. Loading environments that include other modes of deformation, such as lateral translations or rotations, can create a displacement vector that is not aligned with this sensitive axis. To demonstrate this, the dummy chest was loaded quasistatically and dynamically in a series of tests. A string potentiometer array, with the capability to monitor additional deflection modes, was used to supplement the measurement of the chest slider.