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This study presents a detailed comparison of internally and externally measured chest deflections resulting from eight tests conducted on three male post mortem human subjects. A hydraulically driven shoulder belt loaded the anterior thorax under a fixed spine condition while displacement data were obtained via a high-speed 16-camera motion capture system (VICON MX™). Comparison of belt displacement and sternal displacement measured at the bone surface provided a method for quantifying effective change in superficial soft tissue depth at the mid sternum under belt loading. The relationship between the external displacement and the decrease in the effective superficial tissue depth was found to be monotonic and nonlinear. At 65 mm of mid-sternal posterior displacement measured externally, the effective thickness of the superficial tissues and air gap between the belt and the skin had decreased by 14 mm relative to the unloaded state.

A finite element human model has been developed to simulate occupant behavior and to estimate injuries in real-world car crashes. The model represents an average adult male of the US population in a driving posture. Physical geometry, mechanical characteristics and joint structures were replicated as precise as possible. The total number of nodes and materials is around 67,000 and 1,000 respectively. Each part of the model was not only validated against human test data in the literature but also for realistic loading conditions. Additional tests were newly conducted to reproduce realistic loading to human subjects. A data set obtained in human volunteer tests was used for validating the neck part. The head-neck kinematics and responses in low-speed rear impacts were compared between the measured and calculated results. The validity of the lower extremity part was examined by comparing the tibia force in a foot impact between the test data and simulation results.

Current crash dummy biofidelity standards include the estimated effects of tensing the muscles of the thorax. This study reviewed the decision to incorporate muscle tensing by examining relevant past studies and by using an existing mathematical model of thoracic impacts. The study finds evidence that muscle tensing effects are less pronounced than implied by the biofidelity standard response corridors, that the response corridors were improperly modified to include tensing effects, and that tensing of other body regions, such as extremity bracing, may have a much greater effect on the response and injury potential than tensing of only the thoracic musculature. Based on these findings, it is recommended that muscle tensing should be eliminated from thoracic biofidelity requirements until there is sufficient information regarding multi-region muscle tensing response and the capability to incorporate this new data into a crash dummy.

Rupture of the thoracic aorta is a leading cause of rapid fatality in automobile crashes, but the mechanism of this injury remains unknown. One commonly postulated mechanism is a differential motion of the aortic arch relative to the heart and its neighboring vessels caused by high-magnitude acceleration of the thorax. Recent Indy car crash data show, however, that humans can withstand accelerations exceeding 100 g with no injury to the thoracic vasculature. This paper presents a method to investigate the efficacy of acceleration as an aortic injury mechanism using high-acceleration, low chest deflection sled tests. The repeatability and predictability of the test method was evaluated using two Hybrid III tests and two tests with cadaver subjects. The cadaver tests resulted in sustained mid-spine accelerations of up to 80 g for 20 ms with peak mid-spine accelerations of up to 175 g, and maximum chest deflections lower than 11% of the total chest depth.

This paper presents thoracic response corridors developed using fifteen post-mortem human subjects (PMHS) subjected to single and double diagonal belt, distributed, and hub loading on the anterior thorax. We believe this is the first study to quantify the force-deflection response of the same thorax to different loading conditions using dynamic, non-impact, restraint-like loading. Subjects were positioned supine on a table and a hydraulic master-slave cylinder arrangement was used with a high-speed materials testing machine to provide controlled chest deflection at a rate similar to that experienced by restrained PMHS in a 48-km/h sled test. All loading conditions were tested at a nominally non-injurious level initially. When the battery of non-injurious tests was completed, a single loading condition was used for a final, injurious test (nominal 40% chest deflection).

The human body undergoes a variety of changes as it ages through adulthood. These include both morphological (structural) changes (e.g., increased thoracic kyphosis) and material changes (e.g., osteoporosis). The purpose of this study is to evaluate structural changes that occur in the aging bony thorax and to assess the importance of these changes relative to the well-established material changes. The study involved two primary components. First, full-thorax computed tomography (CT) scans of 161 patients, age 18 to 89 years, were analyzed to quantify the angle of the ribs in the sagittal plane. A significant association between the angle of the ribs and age was identified, with the ribs becoming more perpendicular to the spine as age increased (0.08 degrees/year, p=0.012). Next, a finite element model of the thorax was used to evaluate the importance of this rib angle change relative to other factors associated with aging.

This paper describes the injuries generated during dynamic belt loading to a porcine model of the 6-year-old human abdomen, and correlates injury outcomes with measurable parameters. The test fixture produced transverse, dynamic belt loading on the abdomen of 47 immediately post-mortem juvenile swine at two locations (upper/lower), with penetration magnitudes ranging from 23% – 65% of the undeformed abdominal depth, with and without muscle tensing, and over a belt penetration rate range of 2.9 m/s – 7.8 m/s. All thoracoabdominal injuries were documented in detail and then coded according to the Abbreviated Injury Scale (AIS). Observed injuries ranged from AIS 1 to AIS 4. The injury distribution matched well the pattern of injuries observed in a large sample of children exposed to seatbelt loading in the field, with most of the injuries in the lower abdomen.

The abdomen is the second most commonly injured region in children using adult seat belts, but engineers are limited in their efforts to design systems that mitigate these injuries since no current pediatric dummy has the capability to quantify injury risk from loading to the abdomen. This paper develops a porcine (sus scrofa domestica) model of the 6-year-old human's abdomen, and then defines the biomechanical response of this abdominal model. First, a detailed abdominal necropsy study was undertaken, which involved collecting a series of anthropometric measurements and organ masses on 25 swine, ranging in age from 14 to 429 days (4-101 kg mass). These were then compared to the corresponding human quantities to identify the best porcine representation of a 6-year-old human's abdomen. This was determined to be a pig of age 77 days, and whole-body mass of 21.4 kg.

Traumatic aortic rupture (TAR) accounts for a significant mortality in automobile crashes. A numerical method by means of a mesh-based code coupling is employed to elucidate the injury mechanism of TAR. The aorta is modeled as a single-layered thick wall composed of two families of collagen fibers using an anisotropic strain energy function with consideration of viscoelasticity. A set of constitutive parameters is identified from experimental data of the human aorta, providing strict local convexity. An in vitro aorta model reconstructed from the Visible Human dataset is applied to the pulsatile blood flow to establish the references of mechanical quantities for physiological conditions. A series of simulations is performed using the parameterized impact pulses obtained from frontal sled tests.

Very little experimental research has focused on the kinematics, dynamics, and injuries of rear-seated occupants. This study seeks to develop a baseline response for rear-seated post mortem human surrogates (PMHS) in frontal crashes. Three PMHS sled tests were performed in a sled buck designed to represent the interior rear-seat compartment of a contemporary midsized sedan. All occupants were positioned in the right-rear passenger seat and subjected to simulated frontal crashes with an impact speed of 48 km/h. The subjects were restrained by a standard, rear seat, 3-point seat belt. The response of each subject was evaluated in terms of whole-body kinematics, dynamics, and injury. All the PMHS experienced excessive forward translation of the pelvis resulting in a backward rotation of the torso at the time of maximum forward excursion.

This study investigates the performance of a 3-point restraint system incorporating an inflatable shoulder belt with a nominal 2.5-kN load limiter and a non-inflatable lap belt with a pretensioner (the “Airbelt”). Frontal impacts with PMHS in a rear seat environment are presented and the Airbelt system is contrasted with an earlier 3-point system with inflatable lap and shoulder belts but no load-limiter or pretensioners, which was evaluated with human volunteers in the 1970s but not fully reported in the open literature (the “Inflataband”). Key differences between the systems include downward pelvic motion and torso recline with the Inflataband, while the pelvis moved almost horizontally and the torso pitched forward with the Airbelt. One result of these kinematic differences was an overall more biomechanically favorable restraint loading but greater maximum forward head excursion with the Airbelt.

The literature contains a wide range of response data describing the biomechanics of isolated body regions. Current data for the validation of frontal anthropomorphic test devices and human body computational models lack, however, a detailed description of the whole-body response to loading with contemporary restraints in automobile crashes.

Thoracic trauma is the principle causative factor in 30% of road traffic deaths. Researchers have developed force-deflection corridors of the thorax for various loading conditions in order to elucidate injury mechanisms and to validate the mechanical response of ATDs and numerical human models. A corridor, rather than a single response characteristic, results from the variability inherent in biological experimentation. This response variability is caused by both intrinsic and extrinsic factors. The intrinsic factors are associated with individual differences among human subjects, e.g., the differences in material properties and in body geometry. The extrinsic sources of variability include fluctuations in the loading and supporting conditions in experimental tests.