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As part of a continuing study of thoracic injury resulting from blunt frontal loading, the interrelationship of velocity and chest compression was investigated in a series of animal experiments. Anesthetized male swine were suspended in their natural posture and subjected to midsternal, ventrodorsad impact. Twelve animals were struck at a velocity of 14.5 ± 0.9 m/s and experienced a controlled thoracic compression of either 15, 19, or 24%. Six others were impacted at 9.7 ± 1.3 m/s with a greater mean compression of 27%. For the 14.5 m/s exposures the severity of trauma increased with increasing compression, ranging from minor to fatal. Injuries included skeletal fractures, pulmonary contusions, and cardiovascular ruptures leading to tamponade and hemothorax. Serious cardiac arrhythmias also occurred, including one case of lethal ventricular fibrillation. The 9.7 m/s exposures produced mainly pulmonary contusion, ranging in severity from moderate to critical.

The influence of body posture, and inherently support, on thoracic impact response was investigated in an animal model. Anesthetized and postmortem domestic swine were exposed to blunt, midsternal loading while supported in their natural quadrupedal posture, and the results were compared with previously reported data from similar tests involving an upright body orientation. Twelve male animals were tested, six while anesthetized and six postmortem. Each animal was impacted once by a 21 kg rigid mass with a flat contact interface moving at a nominal velocity of either 8 or 10 m/s. Measured mechanical responses included applied load, sternal and spinal accelerations, thoracic compression and aortic overpressure. Injury response was assessed from a thoracico-abdominal necropsy. In addition, ECG traces were recorded pre and postimpact to monitor electro-physiological response.

Five anesthetized porcine subjects were exposed to blunt thoracic impact using a 21 kg mass with a flat contact surface traveling at 3.0 to 12.2 m/s. The experiments were conducted to assess the appropriateness of studying in vivo mechanical and physiological response to thoracic impact in a porcine animal model. A comprehensive review of comparative anatomy between the pig and man indicates that the cardiovascular, respiratory and thoracic skeletal systems of the pig are anatomically and functionally a good parallel of similar structures in man. Thoracic anthropometry measurements document that the chest of a 50 to 60 kg pig is similar to the 50th percentile adult male human, but is narrower and deeper. Peak applied force and chest deflection are in good agreement between the animal's responses and similar impact severity data on fresh cadavers.

Results indicate the need for a redesigned Hybrid III face capable of accurate force and acceleration measurements. New instrumentation and methods for facial fracture detection were developed, including the application of acoustic emissions. Force/ deflection information for the human cadaver head and the Hybrid III ATD were generated for the frontal, zygomatic, and maxillary regions.

Results of two studies concerning the interrelationship of velocity, compression and injury in blunt thoracic impact to anesthetized swine have been combined to provide a data base of forty-one experiments. impact velocity ranged from ∼8-30 m/s and applied normalized chest compression from ∼0.10-0.30. Experimental subjects were suspended in the spine-horizontal position and loaded midsternally through a 150 mm diameter, flat rigid disk on an impacting mass propelled upward from below. Measurements and computations included sternal and spinal accelerations, intracardiovascular overpressures, physiological responses, injury, as assessed by necropsy, and different forms of the velocity and compression exposure severity parameters. The significance of both compression and velocity as parameters of impact exposure severity is clearly demonstrated. Qualitatively, exacerbation of injury was seen when either variable was increased with the other held constant.