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Rupture of the thoracic aorta is a leading cause of rapid fatality in automobile crashes, but the mechanism of this injury remains unknown. One commonly postulated mechanism is a differential motion of the aortic arch relative to the heart and its neighboring vessels caused by high-magnitude acceleration of the thorax. Recent Indy car crash data show, however, that humans can withstand accelerations exceeding 100 g with no injury to the thoracic vasculature. This paper presents a method to investigate the efficacy of acceleration as an aortic injury mechanism using high-acceleration, low chest deflection sled tests. The repeatability and predictability of the test method was evaluated using two Hybrid III tests and two tests with cadaver subjects. The cadaver tests resulted in sustained mid-spine accelerations of up to 80 g for 20 ms with peak mid-spine accelerations of up to 175 g, and maximum chest deflections lower than 11% of the total chest depth.

The human body undergoes a variety of changes as it ages through adulthood. These include both morphological (structural) changes (e.g., increased thoracic kyphosis) and material changes (e.g., osteoporosis). The purpose of this study is to evaluate structural changes that occur in the aging bony thorax and to assess the importance of these changes relative to the well-established material changes. The study involved two primary components. First, full-thorax computed tomography (CT) scans of 161 patients, age 18 to 89 years, were analyzed to quantify the angle of the ribs in the sagittal plane. A significant association between the angle of the ribs and age was identified, with the ribs becoming more perpendicular to the spine as age increased (0.08 degrees/year, p=0.012). Next, a finite element model of the thorax was used to evaluate the importance of this rib angle change relative to other factors associated with aging.

Traumatic aortic rupture (TAR) accounts for a significant mortality in automobile crashes. A numerical method by means of a mesh-based code coupling is employed to elucidate the injury mechanism of TAR. The aorta is modeled as a single-layered thick wall composed of two families of collagen fibers using an anisotropic strain energy function with consideration of viscoelasticity. A set of constitutive parameters is identified from experimental data of the human aorta, providing strict local convexity. An in vitro aorta model reconstructed from the Visible Human dataset is applied to the pulsatile blood flow to establish the references of mechanical quantities for physiological conditions. A series of simulations is performed using the parameterized impact pulses obtained from frontal sled tests.

Very little experimental research has focused on the kinematics, dynamics, and injuries of rear-seated occupants. This study seeks to develop a baseline response for rear-seated post mortem human surrogates (PMHS) in frontal crashes. Three PMHS sled tests were performed in a sled buck designed to represent the interior rear-seat compartment of a contemporary midsized sedan. All occupants were positioned in the right-rear passenger seat and subjected to simulated frontal crashes with an impact speed of 48 km/h. The subjects were restrained by a standard, rear seat, 3-point seat belt. The response of each subject was evaluated in terms of whole-body kinematics, dynamics, and injury. All the PMHS experienced excessive forward translation of the pelvis resulting in a backward rotation of the torso at the time of maximum forward excursion.

Rear seat adult occupant protection is receiving increased attention from the automotive safety community. Recent anthropomorphic test device (ATD) studies have suggested that it may be possible to improve kinematics and reduce injuries to rear seat occupants in frontal collisions by incorporating shoulder-belt force-limiting and pretensioning (FL+PT) technologies into rear seat 3-point belt restraints. This study seeks to further investigate the feasibility and potential kinematic benefits of a FL+PT rear seat, 3-point belt restraint system in a series of 48 kmh frontal impact sled tests (20 g, 80 ms sled acceleration pulse) performed with post mortem human surrogates (PMHS). Three PMHS were tested with a 3-point belt restraint with a progressive (two-stage) force limiting and pretensioning retractor in a sled buck representing the rear seat occupant environment of a 2004 mid-sized sedan.

Thoracic trauma is the principle causative factor in 30% of road traffic deaths. Researchers have developed force-deflection corridors of the thorax for various loading conditions in order to elucidate injury mechanisms and to validate the mechanical response of ATDs and numerical human models. A corridor, rather than a single response characteristic, results from the variability inherent in biological experimentation. This response variability is caused by both intrinsic and extrinsic factors. The intrinsic factors are associated with individual differences among human subjects, e.g., the differences in material properties and in body geometry. The extrinsic sources of variability include fluctuations in the loading and supporting conditions in experimental tests.