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This paper discusses struck-side occupant thoracic response to side-impact loading and the requirements of a sled system capable of reproducing the relevant motions of a laterally impacted vehicle. A simplified viscoelastic representation of a thorax is used to evaluate the effect of the door velocity-time profile on injury criteria and on the internal stress state of the thorax. Simulations using a prescribed door velocity-time profile (punch impact) are contrasted against simulations using a constant-velocity impact (Heidelberg-type impact). It is found that the stress distribution and magnitude within the thorax, in addition to the maximum thorax compression and viscous response, depend not only on the door-occupant closing velocity, but also on the shape of the door velocity-time profile throughout the time of contact with the occupant. A sled system capable of properly reproducing side-impact door and seat motion is described.

This paper presents a computational study of the effects of three parameters on the resulting thoracic injury criteria in side impacts. The parameters evaluated are a) door velocity-time (V-t) profile, b) door interior padding modulus, and c) initial door-to-occupant offset. Regardless of pad modulus, initial offset, or the criterion used to assess injury, higher peak door velocity is shown to correspond with more severe injury. Injury outcome is not, however, found to be sensitive to the door velocity at the time of first occupant contact. A larger initial offset generally is found to result in lower injury, even when the larger offset results in a higher door velocity at occupant contact, because the increased offset results in contact later in the door V-t profile - closer to the point at which the door velocity begins to decrease. Cases of contradictory injury criteria trends are identified, particularly in response to changes in the pad modulus.

The University of Virginia is investigating the biomechanical response and the injury tolerance of the lower extremities. This paper presents the experimental and simulation work used to study the injury patterns and mechanisms of the ankle/foot complex. The simulation effort has developed a segmented lower limb and foot model for an occupant simulator program to study the interactions of the foot with intruding toepan and pedal components. The experimental procedures include static tests, pendulum impacts, and full-scale sled tests with the Advanced Anthropomorphic Test Device and human cadavers. In these tests, the response of the lower extremities is characterized with analogous dummy and cadaver instrumentation packages that include strain gauges, electrogoniometers, angular rate sensors, accelerometers, and load cells. An external apparatus is applied to the surrogate's lower extremities to simulate the effects of muscle tensing.

Rupture of the thoracic aorta is a leading cause of rapid fatality in automobile crashes, but the mechanism of this injury remains unknown. One commonly postulated mechanism is a differential motion of the aortic arch relative to the heart and its neighboring vessels caused by high-magnitude acceleration of the thorax. Recent Indy car crash data show, however, that humans can withstand accelerations exceeding 100 g with no injury to the thoracic vasculature. This paper presents a method to investigate the efficacy of acceleration as an aortic injury mechanism using high-acceleration, low chest deflection sled tests. The repeatability and predictability of the test method was evaluated using two Hybrid III tests and two tests with cadaver subjects. The cadaver tests resulted in sustained mid-spine accelerations of up to 80 g for 20 ms with peak mid-spine accelerations of up to 175 g, and maximum chest deflections lower than 11% of the total chest depth.

This paper describes the injuries generated during dynamic belt loading to a porcine model of the 6-year-old human abdomen, and correlates injury outcomes with measurable parameters. The test fixture produced transverse, dynamic belt loading on the abdomen of 47 immediately post-mortem juvenile swine at two locations (upper/lower), with penetration magnitudes ranging from 23% – 65% of the undeformed abdominal depth, with and without muscle tensing, and over a belt penetration rate range of 2.9 m/s – 7.8 m/s. All thoracoabdominal injuries were documented in detail and then coded according to the Abbreviated Injury Scale (AIS). Observed injuries ranged from AIS 1 to AIS 4. The injury distribution matched well the pattern of injuries observed in a large sample of children exposed to seatbelt loading in the field, with most of the injuries in the lower abdomen.

Injury to the thorax is the predominant cause of fatalities in crash-involved automobile occupants over the age of 65, and many elderly-occupant automobile fatalities occur in crashes below compliance or consumer information test speeds. As the average age of the automotive population increases, thoracic injury prevention in lower severity crashes will play an increasingly important role in automobile safety. This study presents the results of a series of sled tests to investigate the thoracic deformation, kinematic, and injury responses of belted post-mortem human surrogates (PMHS, average age 44 years) and frontal anthropomorphic test devices (ATDs) in low-speed frontal crashes. Nine 29 km/h (three PMHS, three Hybrid III 50th% male ATD, three THOR-NT ATD) and three 38 km/h (one PMHS, two Hybrid III) frontal sled tests were performed to simulate an occupant seated in the right front passenger seat of a mid-sized sedan restrained with a standard (not force-limited) 3-point seatbelt.

Very little experimental research has focused on the kinematics, dynamics, and injuries of rear-seated occupants. This study seeks to develop a baseline response for rear-seated post mortem human surrogates (PMHS) in frontal crashes. Three PMHS sled tests were performed in a sled buck designed to represent the interior rear-seat compartment of a contemporary midsized sedan. All occupants were positioned in the right-rear passenger seat and subjected to simulated frontal crashes with an impact speed of 48 km/h. The subjects were restrained by a standard, rear seat, 3-point seat belt. The response of each subject was evaluated in terms of whole-body kinematics, dynamics, and injury. All the PMHS experienced excessive forward translation of the pelvis resulting in a backward rotation of the torso at the time of maximum forward excursion.