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This study evaluated the response of restrained post-mortem human subjects (PMHS) in 40 km/h frontal sled tests. Eight male PMHS were restrained on a rigid planar seat by a custom 3-point shoulder and lap belt. A video motion tracking system measured three-dimensional trajectories of multiple skeletal sites on the torso allowing quantification of ribcage deformation. Anterior and superior displacement of the lower ribcage may have contributed to sternal fractures occurring early in the event, at displacement levels below those typically considered injurious, suggesting that fracture risk is not fully described by traditional definitions of chest deformation. The methodology presented here produced novel kinematic data that will be useful in developing biofidelic human models.

The abdomen is the second most commonly injured region in children using adult seat belts, but engineers are limited in their efforts to design systems that mitigate these injuries since no current pediatric dummy has the capability to quantify injury risk from loading to the abdomen. This paper develops a porcine (sus scrofa domestica) model of the 6-year-old human's abdomen, and then defines the biomechanical response of this abdominal model. First, a detailed abdominal necropsy study was undertaken, which involved collecting a series of anthropometric measurements and organ masses on 25 swine, ranging in age from 14 to 429 days (4-101 kg mass). These were then compared to the corresponding human quantities to identify the best porcine representation of a 6-year-old human's abdomen. This was determined to be a pig of age 77 days, and whole-body mass of 21.4 kg.

Thoracic trauma is the principle causative factor in 30% of road traffic deaths. Researchers have developed force-deflection corridors of the thorax for various loading conditions in order to elucidate injury mechanisms and to validate the mechanical response of ATDs and numerical human models. A corridor, rather than a single response characteristic, results from the variability inherent in biological experimentation. This response variability is caused by both intrinsic and extrinsic factors. The intrinsic factors are associated with individual differences among human subjects, e.g., the differences in material properties and in body geometry. The extrinsic sources of variability include fluctuations in the loading and supporting conditions in experimental tests.

Current crash dummy biofidelity standards include the estimated effects of tensing the muscles of the thorax. This study reviewed the decision to incorporate muscle tensing by examining relevant past studies and by using an existing mathematical model of thoracic impacts. The study finds evidence that muscle tensing effects are less pronounced than implied by the biofidelity standard response corridors, that the response corridors were improperly modified to include tensing effects, and that tensing of other body regions, such as extremity bracing, may have a much greater effect on the response and injury potential than tensing of only the thoracic musculature. Based on these findings, it is recommended that muscle tensing should be eliminated from thoracic biofidelity requirements until there is sufficient information regarding multi-region muscle tensing response and the capability to incorporate this new data into a crash dummy.

Concern about crash conditions other than frontal and side crashes has accelerated restraint development with respect to rollover events. Previous analysis of rollover field data indicates the high probability of ejection and consequent serious injury or death to unbelted occupants. Partial ejection of belted occupants may also occur. Restraint development has focused on belt technologies and more recently, airbag systems as a method to reduce ejection and injury risk. Effective restraint development for these emerging technologies should consider a combined approach of field injury data analysis, computer simulation of rollover, corresponding validated test data and hardware development techniques. First, crash data was analyzed for identified rollover modes (crash sequences) and injured body regions. This helped to determine possible restraint interventions.

The crash modes that occur each day on streets and highways have not changed dramatically over the past 50 years. The need to better understand those crash modes and their relation to rapidly emerging, tailorable restraint systems has intensified recently. The algorithms necessary for predicting a deployment event are based on an approach of coupling the occupant kinematics in a crash to the sensing technology that will activate the restraint system. This paper describes methods of computer modeling, occupant sensing and vehicle crash dynamics to define a crash sensing system that reacts to a complex set of input conditions to invoke an effective restraint response.

Considerable research has shown that there are two mechanisms of blunt injury. One is by crushing the tissue at low velocities of deformation (compression mechanism, C) and the other by a rate-dependent deformation at higher speeds that exceed the energy dissipation of the tissue (viscous mechanism, VC). Analysis of injury causation in experiments must consider both mechanisms. For an impact, there is a peak compression and Viscous response; however, it is not possible a priori to determine which mechanism is associated with the injury. Thus, there has been a need to identify the effective velocity separating the two mechanisms of injury. This study provides new injury tolerances and probability functions for various body and tissue impacts based on injury data related to a compression or viscous mechanism. Six data sets were subjected to statistical analysis to predict injury based on maximum compression and Viscous response of the surrogate or tissue.