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Rupture of the thoracic aorta is a leading cause of rapid fatality in automobile crashes, but the mechanism of this injury remains unknown. One commonly postulated mechanism is a differential motion of the aortic arch relative to the heart and its neighboring vessels caused by high-magnitude acceleration of the thorax. Recent Indy car crash data show, however, that humans can withstand accelerations exceeding 100 g with no injury to the thoracic vasculature. This paper presents a method to investigate the efficacy of acceleration as an aortic injury mechanism using high-acceleration, low chest deflection sled tests. The repeatability and predictability of the test method was evaluated using two Hybrid III tests and two tests with cadaver subjects. The cadaver tests resulted in sustained mid-spine accelerations of up to 80 g for 20 ms with peak mid-spine accelerations of up to 175 g, and maximum chest deflections lower than 11% of the total chest depth.

A finite element human model has been developed to simulate occupant behavior and to estimate injuries in real-world car crashes. The model represents an average adult male of the US population in a driving posture. Physical geometry, mechanical characteristics and joint structures were replicated as precise as possible. The total number of nodes and materials is around 67,000 and 1,000 respectively. Each part of the model was not only validated against human test data in the literature but also for realistic loading conditions. Additional tests were newly conducted to reproduce realistic loading to human subjects. A data set obtained in human volunteer tests was used for validating the neck part. The head-neck kinematics and responses in low-speed rear impacts were compared between the measured and calculated results. The validity of the lower extremity part was examined by comparing the tibia force in a foot impact between the test data and simulation results.

This study presents a detailed comparison of internally and externally measured chest deflections resulting from eight tests conducted on three male post mortem human subjects. A hydraulically driven shoulder belt loaded the anterior thorax under a fixed spine condition while displacement data were obtained via a high-speed 16-camera motion capture system (VICON MX™). Comparison of belt displacement and sternal displacement measured at the bone surface provided a method for quantifying effective change in superficial soft tissue depth at the mid sternum under belt loading. The relationship between the external displacement and the decrease in the effective superficial tissue depth was found to be monotonic and nonlinear. At 65 mm of mid-sternal posterior displacement measured externally, the effective thickness of the superficial tissues and air gap between the belt and the skin had decreased by 14 mm relative to the unloaded state.