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Rupture of the thoracic aorta is a leading cause of rapid fatality in automobile crashes, but the mechanism of this injury remains unknown. One commonly postulated mechanism is a differential motion of the aortic arch relative to the heart and its neighboring vessels caused by high-magnitude acceleration of the thorax. Recent Indy car crash data show, however, that humans can withstand accelerations exceeding 100 g with no injury to the thoracic vasculature. This paper presents a method to investigate the efficacy of acceleration as an aortic injury mechanism using high-acceleration, low chest deflection sled tests. The repeatability and predictability of the test method was evaluated using two Hybrid III tests and two tests with cadaver subjects. The cadaver tests resulted in sustained mid-spine accelerations of up to 80 g for 20 ms with peak mid-spine accelerations of up to 175 g, and maximum chest deflections lower than 11% of the total chest depth.

This study evaluated the response of restrained post-mortem human subjects (PMHS) in 40 km/h frontal sled tests. Eight male PMHS were restrained on a rigid planar seat by a custom 3-point shoulder and lap belt. A video motion tracking system measured three-dimensional trajectories of multiple skeletal sites on the torso allowing quantification of ribcage deformation. Anterior and superior displacement of the lower ribcage may have contributed to sternal fractures occurring early in the event, at displacement levels below those typically considered injurious, suggesting that fracture risk is not fully described by traditional definitions of chest deformation. The methodology presented here produced novel kinematic data that will be useful in developing biofidelic human models.

Pedestrian impact protection has been a growing area of research over the past twenty or more years. The results from many studies have shown the importance of providing protection to vulnerable road users as a means of reducing roadway fatalities. Most of this research has focused on the vehicle fleet as a whole in datasets that are dominated by passenger cars (cars). Historically, the influence of vehicle body type on injury distribution patterns for pedestrians has not been a primary research focus. In this study we used the Pedestrian Crash Data Study (PCDS) database of detailed pedestrian crash investigations to identify how injury patterns differ for pedestrians struck by light trucks, vans, and sport utility vehicles (LTVs) from those struck by cars. AIS 2+ and 3+ injuries for each segment of vehicles were mapped back to both the body region of the pedestrian injured and the vehicle source linked to that injury in the PCDS database.